By Lubo Zhang, Charles A. Ducsay
To have a good time the heart for Perinatal Biology’s fortieth Anniversary, an illustrious staff amassed at Loma Linda college in February 2013. That accumulating of specialists and this quantity of the court cases are a tribute to the founding father of the heart, Lawrence D. Longo, M.D. those chapters current contributions from people who not directly or one other have been stimulated by way of Dr. Longo. overlaying a variety of subject matters, and illustrating the range of considering and medical pursuits, those lawsuits handle easy technology via to medical difficulties within the developmental programming of future health and illness.
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Extra info for Advances in Fetal and Neonatal Physiology: Proceedings of the Center for Perinatal Biology 40th Anniversary Symposium
10. Janssens SP, Shimouchi A, Quertermous T, Bloch DB, Bloch KD. Cloning and expression of a cDNA encoding human endothelium-derived relaxing factor/nitric oxide synthase. J Biol Chem. 1992;267:14519–22. 11. Marsden PA, Schappert KT, Chen HS, Flowers M, Sundell CL, Wilcox JN, et al. Molecular cloning and characterization of human endothelial nitric oxide synthase. FEBS Lett. 1992;307:287–93. 12. Sessa WC, Harrison JK, Barber CM, Zeng D, Durieux ME, D’Angelo DD, et al. Molecular cloning and expression of a cDNA encoding endothelial cell nitric oxide synthase.
VI Reunion ALIRH; 1974. p. 89. 6. Garcia-Austt J, Alonso J, Loayza A, Oddone R, Sabatino H, Caldeyro-Barcia R. Comparación de los Efectos Cardiovasculares Fetales en Oclusiones de Vasos Umbilicales y de Aorta Materna en Fetos de Oveja. VI Congreso Uruguayo de Ginecotocología, vol. 2, Montevideo; 1974. pp. 503–7. 7. Loayza A, Garcia-Austt J, Alonso J, Carmona O, Sabatino H, Caldeyro-Barcia R. Comparacion de las Modificaciones de la PO2 y PCO2 en Sangre Fetal en Oclusiones de Vasos Umbilicales y de Aorta Materna en Ovejas.
But to get there we need to know two things: (1) How is pregnancy-adapted endothelial vasodilatory function achieved in the first place, and (2) Could knowing the molecular cause of endothelial failure also help identify a potential drug target to restore function in human disease? A number of studies in intact rat and sheep uterine artery have answered the first question, revealing that it is pregnancy-specific augmentation of sustained endothelial Ca2+ signaling that underlies pregnancy-enhanced NO production and corresponding increases in vasodilation [3–6].
Advances in Fetal and Neonatal Physiology: Proceedings of the Center for Perinatal Biology 40th Anniversary Symposium by Lubo Zhang, Charles A. Ducsay