By C Alzheimer
Read Online or Download Molecular and Cellular Biology of Neuroprotection in the CNS PDF
Similar biology books
A set of state-of-the-art suggestions for detecting and quantifying apoptosis, realizing its biochemistry, and for choosing the genes and proteins that control and hold it out. defined in step by step aspect, those without problems reproducible tools variety from move cytometry and immunohistochemical tactics to kinase task assays, yeast two-hybrid screening, and the cloning of novel genes by means of differential expression.
With bankruptcy contributions from greater than 30 steel biology specialists, mobile and Molecular Biology of Metals explains the position of key divalent steel ions thinking about the molecular and mobile biology of varied aim cellphone populations. even though it essentially makes a speciality of homeostatic metals, reminiscent of nickel, zinc, and chromium, the textual content additionally discusses a number of environmentally pertinent, poisonous divalent cations, together with mercury, cadmium, and arsenic.
The time period cytoskeleton has develop into firmly confirmed in trendy clinical vocabulary. certainly, it truly is tricky to think that in simple terms ten years in the past, it used to be nearly non-existent. given that then, the trendy box of analysis at the structural association of the cytoplasm has become probably the most efficient and speedily increasing study components in telephone Biology this day.
- The Ethics of Research Biobanking
- Biology in Stem Cell Niche
- Biology of Fresh Waters
- Life Out of Sequence: A Data-Driven History of Bioinformatics
Extra info for Molecular and Cellular Biology of Neuroprotection in the CNS
Pharmacology of glutamate neurotoxicity in cortical cell culture: attenuation by NMDA antagonists. J Neurosci 1988; 8(1):185-196. 20. Koh JY, Goldberg MP, Hartley DM et al. Non-NMDA receptor-mediated neurotoxicity in cortical culture. J Neurosci 1990; 10693-705. 21. Michaels RL, Rothman SM. Glutamate neurotoxicity in vitro: Antagonist pharmacology and intracellular calcium concentrations. J Neurosci 1990; 10(1):283-292. 22. Frandsen A, Drejer J, Schousboe A. Direct evidence that excitotoxicity in cultured neurons is mediated via N-methyl-D-aspartate (NMDA) as well as non-NMDA receptors.
J Cereb Blood Flow Metab 1998; 18(3):238-247. 95. Chen J, Nagayama T, Jin K et al. Induction of caspase-3-like protease may mediate delayed neuronal death in the hippocampus after transient cerebral ischemia. J Neurosci 1998; 18(13):4914-4928. 30 T. GILLESSEN ET AL. 96. Kaul M, Garden GA, Lipton SA. Pathways to neuronal injury and apoptosis in HIV-associated dementia. Nature 2001; 410(6831):988-994. 97. Bezzi P, Domercq M, Brambilla L et al. CXCR4-activated astrocyte glutamate release via TNFalpha: amplification by microglia triggers neurotoxicity.
107. Ross SM, Seelig M, Spencer PS. Specific antagonism of excitotoxic action of ‘uncommon’ amino acids assayed in organotypic mouse cortical cultures. Brain Res 1987; 425(1):120-127. 108. Ross SM, Spencer PS. Specific antagonism of behavioral action of “uncommon” amino acids linked to motor-system diseases. Synapse 1987; 1(3):248-253. 109. Bridges RJ, Stevens DR, Kahle JS et al. Structure-function studies on N-oxalyldiamino-dicarboxylic acids and excitatory amino acid receptors: evidence that beta-L-ODAP is a selective non-NMDA agonist.
Molecular and Cellular Biology of Neuroprotection in the CNS by C Alzheimer