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Pharmacology of glutamate neurotoxicity in cortical cell culture: attenuation by NMDA antagonists. J Neurosci 1988; 8(1):185-196. 20. Koh JY, Goldberg MP, Hartley DM et al. Non-NMDA receptor-mediated neurotoxicity in cortical culture. J Neurosci 1990; 10693-705. 21. Michaels RL, Rothman SM. Glutamate neurotoxicity in vitro: Antagonist pharmacology and intracellular calcium concentrations. J Neurosci 1990; 10(1):283-292. 22. Frandsen A, Drejer J, Schousboe A. Direct evidence that excitotoxicity in cultured neurons is mediated via N-methyl-D-aspartate (NMDA) as well as non-NMDA receptors.

J Cereb Blood Flow Metab 1998; 18(3):238-247. 95. Chen J, Nagayama T, Jin K et al. Induction of caspase-3-like protease may mediate delayed neuronal death in the hippocampus after transient cerebral ischemia. J Neurosci 1998; 18(13):4914-4928. 30 T. GILLESSEN ET AL. 96. Kaul M, Garden GA, Lipton SA. Pathways to neuronal injury and apoptosis in HIV-associated dementia. Nature 2001; 410(6831):988-994. 97. Bezzi P, Domercq M, Brambilla L et al. CXCR4-activated astrocyte glutamate release via TNFalpha: amplification by microglia triggers neurotoxicity.

107. Ross SM, Seelig M, Spencer PS. Specific antagonism of excitotoxic action of ‘uncommon’ amino acids assayed in organotypic mouse cortical cultures. Brain Res 1987; 425(1):120-127. 108. Ross SM, Spencer PS. Specific antagonism of behavioral action of “uncommon” amino acids linked to motor-system diseases. Synapse 1987; 1(3):248-253. 109. Bridges RJ, Stevens DR, Kahle JS et al. Structure-function studies on N-oxalyldiamino-dicarboxylic acids and excitatory amino acid receptors: evidence that beta-L-ODAP is a selective non-NMDA agonist.

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Molecular and Cellular Biology of Neuroprotection in the CNS by C Alzheimer


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